1.92 CME

Pendekatan terhadap Penyakit Paru Obstruktif

Pembicara: Dr. Neel Thakkar

Konsultan, Kedokteran Paru dan Perawatan Intensif, Sterling Hospitals, Vadodara

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Keterangan

Smoking is the main cause of chronic obstructive pulmonary disease, a prevalent systemic illness. Spirometry should be incorporated into primary care practice since it is crucial for the diagnosis of COPD. Both pharmaceutical and nonpharmacologic treatments enhance quality of life, exercise tolerance, and symptom relief. The only strategy that has been proven to reduce the progression of the disease is quitting smoking. To improve health and quality of life, it is necessary to recognize and treat the systemic symptoms and comorbidities linked to COPD.

Ringkasan

  • The session focuses on obstructive lung diseases, emphasizing the relevance of COPD and asthma, especially given the poor air quality in India. It notes that November is World COPD Awareness Month, and air pollution significantly contributes to respiratory disease-related deaths and disabilities. The trachea and bronchioles are discussed, highlighting that pathology can affect both airways and alveoli.
  • Obstructive lung diseases are classified based on reversibility, etiology (allergic, infectious, genetic, environmental), mechanism (airway inflammation, mucus secretion, alveolar destruction), and age of onset. COPD, bronchial asthma, bronchiectasis, cystic fibrosis, and occupational lung diseases are all considered. The session emphasizes the importance of differentiating these conditions.
  • COPD is defined as a heterogeneous lung condition with chronic respiratory symptoms (dyspnea, cough, sputum, exacerbations). Risk factors include smoking, chulha smoking, occupational dust, pollution, genetics, and socioeconomic status. Phenotypes of COPD are identified (G, D, C, P, I, A, U) and clinical indicators for diagnosis (dyspnea, wheezing, chronic cough, risk factors) are outlined.
  • The session contrasts asthma and COPD, noting variability in airflow obstruction and symptoms, as well as a potential history of allergies and family history in asthma. Radiology interpretations of chest X-rays for various conditions like bronchiectasis, pulmonary edema, and cavitary lesions are included. Small airway disease pathology in COPD involves inflammation, fibrosis, and hyperinflation.
  • COPD examination findings include wheezing, pursed-lip breathing, barrel chest, prolonged respiratory time, and signs of right heart failure. Oscultatory findings reveal decreased breath sounds and ronchi. Spirometry is vital for diagnosis and assessing disease severity. The GOLD guidelines for COPD severity classification, using FEV1 values, MMRC breathlessness scale, and CAT assessment score, are explained.
  • Management strategies for COPD involve non-pharmacological approaches like smoking cessation, vaccination, and active lifestyle. Pharmacotherapy is tailored based on GOLD group (A, B, E) and eosinophil levels. Non-pharmacologic interventions include pulmonary rehabilitation, nutrition, and physiotherapy. Home oxygen therapy considerations are outlined. Confounders and contributors to COPD diagnosis such as pneumonia, pulmonary embolism, and heart failure are detailed.
  • GINA guidelines define asthma as a heterogeneous disease with chronic airway inflammation and variable respiratory symptoms. Variability and reversibility are key diagnostic features. Asthma phenotypes are described (allergic, non-allergic, cough variant, adult onset, occupational). Overlap of asthma and COPD is discussed, and the hypothesis of one airway one disease is introduced. Key diagnostic criteria for asthma include variability, reversibility, and challenge tests.

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